![]() Attempting to force oneself onto daytime society's schedule with DSPD has been compared to constantly living with jet lag DSPD has been called "social jet lag". sleeping from 4:00 am to 1:00 pm, their sleep is improved and they may not experience excessive daytime sleepiness. If they are allowed to follow their own schedules, e.g. However, they find it very difficult to wake up in time for a typical school or work day. Unless they have another sleep disorder such as sleep apnea in addition to DSPD, patients can sleep well and have a normal need for sleep. Īffected people often report that while they do not get to sleep until the early morning, they do fall asleep around the same time every day. People with DSPD generally fall asleep some hours after midnight and have difficulty waking up in the morning. People with DSPD may improve their quality of life by choosing careers that allow late sleeping times, rather than forcing themselves to follow a conventional 9-to-5 work schedule. A chief difficulty of treating DSPD is in maintaining an earlier schedule after it has been established, as the patient's body has a strong tendency to reset the sleeping schedule to its intrinsic late times. At its most severe and inflexible, DSPD is a disability. DSPD can be treated or helped in some cases by careful daily sleep practices, morning light therapy, evening dark therapy, earlier exercise and meal times, and medications such as aripiprazole, melatonin, and modafinil melatonin is a natural neurohormone partly responsible for the human body clock. However, since many doctors are unfamiliar with the condition, it often goes untreated or is treated inappropriately DSPD is often misdiagnosed as primary insomnia or as a psychiatric condition. It is responsible for 7–13% of patient complaints of chronic insomnia. Weitzman and others at Montefiore Medical Center. History ĭSPD was first formally described in 1981 by Elliot D. There may be a genetic component to the syndrome. Symptom management may be possible with therapeutic drugs such as orexin antagonists or melatonin receptor agonists, as well as regular outdoor exercise. ![]() Researchers have speculated that the lack of exposure to natural sunrise/sunset cycles relates many of the symptoms of these circadian disorders to modern habits of humans spending extended periods indoors, without sunlight exposure and with artificial light. Their results found significant divergence between individuals, with most participants settling upon a rhythm of 30 +/- 4 hours. In one example, several dozen volunteers spent many months underground in a French cave, while researchers monitored their periods of waking and sleeping. The orexin system was identified only in 1998, yet it appears intimately implicated in human sleep-wake systems.Įvidence for the plasticity of human circadian rhythm cycles has been provided by multiple studies. Many insomnia-related disorders can present significantly differently between patients, and circadian rhythm disorders and melatonin related disorders are not well understood by modern medical science. ![]() The diagnosis of this disorder is currently a point of contention among specialists of sleep disorders. The disorder affects the timing of biological rhythms including sleep, peak period of alertness, core body temperature, and hormonal cycles. Medical condition Delayed sleep phase disorderĭelayed sleep–wake phase disorder, delayed sleep phase syndrome, delayed sleep phase type, social jetlagĬomparison of standard (green) and DSPD (blue) circadian rhythmsĭelayed sleep phase disorder ( DSPD), more often known as delayed sleep phase syndrome and also as delayed sleep–wake phase disorder, is the delaying of a person's circadian rhythm (biological clock) compared to those of societal norms. ![]()
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